See Also: striational antibodies(medicine)
Herbal medicine (botanical medicine, herbology, phytomedicine)(health)
Medicine Lodge Memorial Hospital- Medicine Lodge(health)
Antibodies(health)
Ku antibodies(medicine)
P-K antibodies(medicine)
Antibodies(medicine)
HIV antibodies(medicine)
Orthomolecular medicine (orthomolecular nutritional medicine, orthomolecular therapy)(health)
antibodies, blocking(medicine)

c3 cycle (medicine) and striational antibodies (medicine)


c3 cycle (medicine)


c3 cycle
<biochemistry> The part of the photosynthesis process where carbon dioxide is converted into three-carbon compounds, which can then be turned into six-carbon sugars.


striational antibodies (medicine)


striational antibodies
<immunology> Thymoma occur in approximately 15% of patients with myasthenia gravis. Striational antibodies are found in 90% of patients with both myasthenia gravis and thymoma and are present in only 6% of myasthenia gravis patients <40 years of age without thymoma. They are absent in 82-100% of all patients with myasthenia gravis but without thymoma. The absence of striational antibodies argues against a thymoma in a patient with myasthenia gravis.

Elevated values of striational antibodies (predominantly IgM) are found in 25% of patients with rheumatoid arthritis treated with penicillamine. They are also useful when monitoring immunosuppressive therapy for detection of autoimmune complications of bone marrow transplantation including graft-versus-host disease.

Measurement of striational antibodies and acetylcholine receptor antibodies is useful for predicting risk of myasthenia gravis in patients with thymoma and for predicting recurrence of thymoma. Antibodies to titin, which comprise at least some of the striational antibodies which react with the I band near the A-1 junction, are highly associated with the presence of thymoma in myasthenia gravis.

Alpha-smooth muscle (alpha-SM) is also associated with striational antibodies. The alpha-SM actin isoform is expressed normally by vascular SM cells and by stromal fibroblastic cells in pathological conditions leading to fibrosis. Upregulatation of alpha-SM actin in the glomerulus indicates mesangial cell activation and is not always correlated with the degree of glomerulosclerosis. The relatively high incidence for ASA (anti-smooth muscle antibody) suggests that immune-mediated processes might be involved in the etiology of sudden deafness.